Coffee can be a healthy part of nearly any diet.
“Coffee is dangerous and unhealthy.” This is preposterous and untrue for most of us. The reasoning behind this is coffee contains caffeine, and caffeine is dangerous. Most studies today, however, actually show that coffee has health benefits that outweigh the dangers for most of us.
Caffeine should be restricted in anyone with high blood pressure, kidney disease, a history of stroke, or coronary artery disease. I recommend coffee to anyone who does not have a medical reason not to drink it. Why coffee?
Research:
- Coffee is an excellent source of antioxidants[1],[2],[3]. Antioxidants are needed to decrease inflammation. They have been proven to lower the risk of multiple cancers and coronary artery disease. One study looked at antioxidants and their effect on lipid profiles. This study showed that drinking coffee improved blood lipid panels[4].
- Coffee drinkers have a lower risk of dementia and memory loss[5]. There is some proof that coffee helps with Alzheimer’s and dementia associated with Parkinson’s disease[6],[7]. Numerous studies show that coffee drinkers live longer and have a lower risk of many serious diseases.
- Lastly, research from the New England Journal of Medicine shows a decrease in all causes of mortality[8],[9].
The Truth: Despite the perception that coffee is unhealthy, coffee contains antioxidants that help prevent inflammation and disease. As long as you do not have a health condition that precludes caffeine use, it is safe with some moderation of less than 400 mg per day.
The bottom line: Coffee can be a healthy part of any weight loss or weight maintenance diet as long as you avoid creamer and sugar.
References:
- [1]J. BONITA, M. MANDARANO, D. SHUTA, and J. VINSON, “Coffee and cardiovascular disease: In vitro, cellular, animal, and human studies,” Pharmacological Research, pp. 187–198, Mar. 2007, doi: 10.1016/j.phrs.2007.01.006. [Online]. Available: http://dx.doi.org/10.1016/j.phrs.2007.01.006
- [2]A. Svilaas et al., “Intakes of Antioxidants in Coffee, Wine, and Vegetables Are Correlated with Plasma Carotenoids in Humans,” The Journal of Nutrition, pp. 562–567, Mar. 2004, doi: 10.1093/jn/134.3.562. [Online]. Available: http://dx.doi.org/10.1093/jn/134.3.562
- [3]R. Pulido, M. Hernández-García, and F. Saura-Calixto, “Contribution of beverages to the intake of lipophilic and hydrophilic antioxidants in the Spanish diet,” Eur J Clin Nutr, pp. 1275–1282, Sep. 2003, doi: 10.1038/sj.ejcn.1601685. [Online]. Available: http://dx.doi.org/10.1038/sj.ejcn.1601685
- [4]G. M. Agudelo-Ochoa et al., “Coffee Consumption Increases the Antioxidant Capacity of Plasma and Has No Effect on the Lipid Profile or Vascular Function in Healthy Adults in a Randomized Controlled Trial,” The Journal of Nutrition, pp. 524–531, Feb. 2016, doi: 10.3945/jn.115.224774. [Online]. Available: http://dx.doi.org/10.3945/jn.115.224774
- [5]F. Panza et al., “Coffee, tea, and caffeine consumption and prevention of late-life cognitive decline and dementia: A systematic review,” J Nutr Health Aging, pp. 313–328, Dec. 2014, doi: 10.1007/s12603-014-0563-8. [Online]. Available: http://dx.doi.org/10.1007/s12603-014-0563-8
- [6]Y. Wang et al., “Effects of caffeic acid on learning deficits in a model of Alzheimer’s disease,” International Journal of Molecular Medicine, pp. 869–875, Jul. 2016, doi: 10.3892/ijmm.2016.2683. [Online]. Available: http://dx.doi.org/10.3892/ijmm.2016.2683
- [7]M. A. Hernán, B. Takkouche, F. Caamaño-Isorna, and J. J. Gestal-Otero, “A meta-analysis of coffee drinking, cigarette smoking, and the risk of Parkinson’s disease,” Ann Neurol., pp. 276–284, May 2002, doi: 10.1002/ana.10277. [Online]. Available: http://dx.doi.org/10.1002/ana.10277
- [8]N. D. Freedman, Y. Park, C. C. Abnet, A. R. Hollenbeck, and R. Sinha, “Association of Coffee Drinking with Total and Cause-Specific Mortality,” N Engl J Med, pp. 1891–1904, May 2012, doi: 10.1056/nejmoa1112010. [Online]. Available: http://dx.doi.org/10.1056/NEJMoa1112010
- [9]E. Loftfield et al., “Association of Coffee Consumption With Overall and Cause-Specific Mortality in a Large US Prospective Cohort Study,” Am. J. Epidemiol., p. kwv146, Nov. 2015, doi: 10.1093/aje/kwv146. [Online]. Available: http://dx.doi.org/10.1093/aje/kwv146
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